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Monday, September 25, 2006

Good news for Alzheimer disease.



Portrait of an Alzheimer's patient.

"Estamos cerca de descubrir los secretos de la E. de Alzheimer y revertirla".

http://www.youtube.com/watch?v=GYfcKSnXRy0


New therapy in Alzheimer disease.This patient believe the genetic therapy slower down her disease.

2 scientific works related with the illness of Alzheimer: insanity conditioned by deposits of brain cerebral b-amiloyd protein that annihilating nervous cells generates insanity and relentless forgetfulness of all the things, are important. Christian Haas and Michael Willem, biochemists of the University Ludwig/Maximilian of Munich / Germany, have discovered an enzyme: Bace-1 that acting by way of organic scissor cut the b-amiloyd of a protein but big, accumulating it in the the brain, in form of plates. As it is logical to suppose, counteracting the action of the enzyme the effects of Alzheimer would be counteracted. A discovery of relevance, except for a detail. The same enzyme: Bace-1, generates (at least in newly born mice), sheaths of mielyn, around the normal nerves in formation, fundamental for a nervous effective conduction. That's to say, we can stop or to cure Alzheimer, at the same time that we condition parestesias or corporal paralysis. The German biochemists are not discouraged, they assure that therapeutic variants can be gestated, in this respect.

The other news not less important occurs of the work of the German Melanie Meyer Luehmann (University of Tubingen/Germany) et al, who has showed that diluted plates of brain b- amiloyd protein of people with Alzheimer or, precursors of b-amiloyd protein -transplanted to brains of mice- produces also plates of b –amiloyd in the brains of these animals with simptoms similar to the illness of Alzheimer. That`s to say, the determining factors of the illness of Alzheimer are trasmitted, in similar way to the effect exercised by prions (illness of crazy cows). If the amiloyd is transmitted sure, it has an infectious origin. Being infectious, is possible to prevent and to treat it. By the quick thing the Meyer, has stopped the abnormal storing of brain b-amiloyd, by means of technics of inmunodepletion, b-amiloyd protein denaturalization and immunization of the host.

Avances notables en la enfermedad de Alzheimer.

2 trabajos cientificos relacionados con la enfermedad de Alzheimer : demencia condicionada por depósitos de b-amiloide cerebral, que aniquilando celulas nerviosas generan demencia y olvido inexorable de todas las cosas. Christian Haas y Michael Willem, bioquimicos de la Universidad Ludwig/Maximilian de Munich/Alemania, han descubierto una enzima : Bace-1, que actuando a modo de tijera corta el b-amiloide de una proteina mas grande, acumulandola en el cerebro, en forma de placas. Como es logico suponer, contrarrestando la accion de la enzima se contrarrestarian los efectos del Alzheimer. Un hallazgo de relevancia, excepto por un detalle. La misma enzima : Bace-1, genera (al menos en ratones recien nacidos), cubiertas (vainas) de mielina, alrededor de los nervios normales en formacion, fundamentales para una conduccion nerviosa eficáz. Es decir, podemos detener o curar al Alzheimer, al mismo tiempo que condicionamos parestesias o paralisis corporales. Los bioquimicos alemanes no se desaniman, aseguran que pueden gestarse variantes terapeuticas, al respecto.

La otra noticia no menos importante deviene del trabajo de la alemana Melanie Meyer Luehman, (Universidad de Tubingen/Alemania) et al, quien ha demostrado que cumulos de b- amiloide cerebral diluido de personas con Alzheimer o, precursores de proteina b-amiloide -trasplantados a cerebros de ratones- producen depósitos de b –amiloide en los cerebros de estos animales de experimentación con sintomatologia similar a la enfermedad de Alzheimer. Es decir, los condicionantes de la enfermedad de Alzheimer, son trasmitibles, de modo semejante al efecto ejercido por priones (enfermedad de las vacas locas). Si el amiloide es trasmitible, tendria que plantearse un origen infeccioso para esta afección. Siendo una infeccion, es posible prevenirla y tratarla. Por lo pronto la Meyer, esta impidiendo los depositos de b-amiloide cerebral, mediante tecnicas de inmunodeplecion, desnaturalizacion de la proteina b-amiloide e inmunización del huésped.

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